Background
Definition 
Palmar Fibromatosis
Aetiology
AD with variable penetration
Pathogenesis
Murrell's Theory of Pathogenesis
1. Microvascular ischaemia
Palmar Fibromatosis
AD with variable penetration
Murrell's Theory of Pathogenesis
1. Microvascular ischaemia
Groin pain in athletes
- secondary to tear in external oblique fascia
Sports with aggressive adduction
- hockey / soccer
Tears in external oblique fascia
- tend to be central
- around spermatic cord and ilioinguinal nerve
- pain may be from nerve entrapment
Adductor Longus Tear
Radius rotates externally in relation to the ulna
- posterior displacement of the radial head relative to the capitellum
- in flexion
A fixed equinus deformity of the forefoot in relation to the hindfoot
- resulting in an abnormally high arch that fails to flatten with weight bearing
- deformity may be forefoot, hindfoot or combination
Osseous canal between talus and calcaneum
- interosseous talo-calcaneal ligament
- cervical ligament
- joint capsule
- nerve endings / arterial anastomoses
Flat foot / overpronation
Inversion / sprain
Bunion
- medial prominence of head of 1st MT
Hallux Valgus
- medial deviation 1st MT
- lateral deviation of great toe
Metatarsal head
- has 2 grooves separating ridge (cristae)
ITB rubbing on LFC
- long distance runners (slow running more at risk than fast)
- cyclists (seat too high, improper technique)
ITB becomes tight, especially posterior portion
Develop inflammed tissue under ITB
- synovium
- bursa
Overuse
- sudden increase in distance
- hill running
- genu varum
- improper shoe wear
Primary malignancy arising from remnants of the notochord
Rare malignant tumour
Middle-aged adults (50-70)
M>F
In midline in axial skeleton
Base of skull 35%
Vertebrae 15%
- especially cervical
Sacrum 50%
Chondroid Metaplasia of synovium affecting large joints
Nodules of hyaline cartilage
- formed in the subsynovial layer of joint capsules
Rare lesion
Most common in 20's and 30's
Sex: M > F (2:1)
Monoarticular
Virchow's Triad
1. Venous stasis
2. Hypercoagulability
3. Endothelial damage
Starts as platelet nidus at valves
- thrombogenic materials elaborated by platelets
- leads to development of fibrin thrombus
- thrombus grows
Thrombus may
- detach as embolus
- be completely dissolved / recanalise
- organise with valve incompetence