Self limiting syndrome of unknown aetiology
- hip pain associated with osteoporosis of proximal femur
AVN
- AVN of the hip in pregnancy is rare but possible
- TOH tends to be diffuse on MRI, while AVN is localised
- extends to neck and metaphysis
- transient osteoporosis has normal bone scan
Rare
- M: F 3:1
Largely related to degree of displacement
Hawkins Type I
- 0% to 13%
Hawkins Type II
- 20% to 50%
- usually only patchy and not a problem (rarely collapses)
10% of elbow tendonitis
Overuse injury
- poor swing in golf
- poor throwing technique
- overuse of topspin in tennis
- occupational (repetitive hammering / screwing)
Some patients also have lateral epicondylitis
Tenderness CFO
Stimulate pain
- flexion of WJ with fingers resisting
Lateral : Medial 9:1
4th & 5th decades
- M = F
- 75% dominant arm
50% of regular tennis players
- especially > 2 hrs / week
Insertion pathology / Enthesopathy
Over-extension of the elbow with supination / pronation
Lateral epicondyle
- anconeus from posterior face
- ECRB and EDC from anterior face (CEO)
Occurs 10% of supracondylar humeral fractures
- varus malunion
Lateral condyle fracture
- AVN trochlea
Growth arrest of medial aspect physis
- rare
- post traumatic
Rotational force incurred while joint partially flexed & extending
- caught between femoral & tibial condyles
- usually valgus & ER / varus & IR
Patient
Advanced age
Immunosuppression - steroids / Rheumatoid / DM
Malnutrition - Lymphocyte count / Transferrin / Albumin
Vascular disease
Obesity
Poor skin i.e. psoriasis
Previous infection in joint
Infection elsewhere - i.e. UTi
Prolonged hospital admission
Revision surgery
Operative Factors
Preoperative
10% of calcaneus fractures
40% of crush injuries
High index of suspicion / classic signs less reliable
Pain & pain with passive stretch remain the cardinal signs
- pallor, paresthesia, pulselessness, & paralysis occur later or sometimes not at all
- low threshold calcaneal compartment pressures & surgical exploration
A. Ankle Fracture
Types
- Weber A 4%
- Weber C 33%
- Displaced large posterior malleolar
Any OA develops in first 2 years
Causes
- articular damage at time of injury
- non anatomical reconstruction
- complications i.e. infection